Sinus Treatment

Tuesday, October 25, 2011

What is Cavernous Sinus?

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To understand what is the cavernous sinus, one needs to understand the structure of human head to some extent. The cavernous sinus, also known as lateral sellar compartment are located within the human head. A large collection of veins constituted of veins with thin walls create this cavity, which is on one side bordered by the temporal bone and on the other by the sphenoid bone of the skull.

What constitutes the cavernous sinus is something like this. Vertically it comprises of trochlear nerve, maxillary nerve, oculomotor nerve and ophthalmic nerve. Horizontally it is made up of abducens nerve and internal carotid artery as well as sympathetic plexus. And as can be easily guessed, all these nerves give it a very complex and delicate character. So it can be safely argued that what cavernous sinus is, is nothing but a cavity in our skull to hold a disparate number of nerves. In this respect we also find that there is similarity between any other sinus and cavernous sinus by virtue of being a cavity in the skull.

Now coming to the connections of the veins, we find that it has got tributaries from both inferior and superior ophthalmic veins, middle and superior cerebral veins and superior parietal sinus. It is the only such anatomical location in our body where an artery has to travel completely through a structure of veins. So if someone is to ask what is unique about cavernous sinus, then it is undoubtedly this. Though this not to negate its similarities with other sinuses.

Our understanding of what is cavernous sinus gets both enhanced and complicated as well when we take into account the fact that pituitary gland lies nowhere else but very much between the two paired cavernous sinuses. Any abnormal growth of pituitary adenoma when expands in a particular direction where there is least resistance, it causes the compression of cavernous sinus. This compression results in cavernous sinus syndrome because of these tumors and ultimately results in opthalmoplegia which is loss of ophthalmic sensory loss while the compression of maxillary nerve causes maxillary sensory loss.

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Tuesday, January 5, 2010

What is Sinus Bradycardia - Understanding Sinus Bradycardia Better

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What is sinus bradycardia and where does it start from. Sinus bradycardia starts from the sinus node; it is a heart rhythm and has a rate of fewer than 60 beats per minute (bps). Simple words on what is sinus bradycardia they are the sinus beat when a resting heart strikes 60 bps or even less. The sinus rhythms cause a P-wave on the ECG which means it is normal in terms of both amplitude and vector. They are tagged on with QRS complex and T waves.

A normal baby should have a heart beat of 150 bps; Bradycardia decelerates the premature baby's heart to less than 100 beats per minute. Infection, anemia, change in body temperature, hypoglycemia, airway problems, neurological difficulties are caused due to Bradycardia.

What are sinus bradycardia symptoms?

Sinus bradycardia does not normally have any symptoms. But at times there are indications like dizziness, lightheadedness; exercise intolerance, chest pain and succinctness of breath are seen.

The problems that result due to bradycardia are of two types: they are sinatorial node (SA node) disorder and atrioventricular node (AV node) disorder. The former is called sick sinus syndrome and is due to the weakened conduction of the impulse from the sinus node. The later may result due to weakened conduction in the AV node or even underneath in the bundle of HIS. This may be acquired congenitally (disorder by birth). It is found most commonly in elderly patients.

What is sinus bradycardia cure?

Treatment for asymptomatic patients is not signified, but symptomatic patients should be corrected of underlying electrolyte and acid base disorder. Intravenous atropine gives relief temporarily. Previously isoproterenol was used in patients with bradycardia, but due to the increase in risk its usage has been lessened. Temporary pacing also is recommended for patients who are not responsive to atropine although transcutaneous pacing is the primary choice for physicians.

The medication techniques include therapy and supratherapeutic doses of digitalis glycosides, beta-blockers and calcium channel blocking agents. Cardiac drugs are also used.

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